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The removal of all the skin pigment with monobenzone is permanent and vigorous.Sun-safety must be adhered to for life to avoid severe sunburn and melanomas. Descriptions of a disease believed to be vitiligo date back to a passage in the medical text Ebers Papyrus circa 1500 BC in ancient Egypt.The longevity of the repigmentation differed from person to person.By now, several transplantation techniques have been developed, including transplantation of melanocyte precursors derived from hair follicles.Psoralen and ultraviolet A light (PUVA) treatment involves taking a drug that increases the skin's sensitivity to ultraviolet light, then exposing the skin to high doses of UVA light.Treatment is required twice a week for 6–12 months or longer.The area to be treated was then denuded with a dermabrader and the melanocytes graft applied.Between 70 and 85 percent of patients experienced nearly complete repigmentation of their skin.

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Interleukin-1β and interleukin-18 are expressed at high levels in patients with vitiligo. The original protein and sequence is highly conserved in evolution, and is found in humans, chimpanzee, rhesus monkey, and the bush baby.Addison's disease (typically an autoimmune destruction of the adrenal glands) may also be seen in individuals with vitiligo.An ultraviolet light can be used in the early phase of this disease for identification and to determine the effectiveness of treatment.A serious potential side effect involves the risk of developing skin cancer, the same risk as an over-exposure to natural sunlight.Ultraviolet light (UVA) treatments are normally carried out in a hospital clinic.Mentions of whitening of the skin were also present circa 1400 BC in sacred Indian texts such as Atharvaveda as well as Shinto prayers in East Asia circa 1200 BC.

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